A California judge has ruled that businesses like Starbucks that sell coffee in the state need to warn consumers that the drink poses a cancer risk because it contains the chemical acrylamide.
Acrylamide in large quantities could be dangerous, but it's in many foods we consume — and can't be removed from coffee.
The amount in coffee is much lower than the doses shown to cause harm.
Existing research suggests coffee consumption is actually associated with lower risks for cancer and other diseases.
Coffee companies like Starbucks were fighting back against the tentative ruling, but, on May 7, a Los Angeles judge made the ruling final.
Pertussis vaccines do not contain pertussis bacteria. The Tdap and DTaP vaccines that were phased in during the 1990s don’t confer immunity to pertussis bacteria. The vaccines contain pertussis toxoid. Toxoid is the inactivated version of the nasty exotoxin that the bacteria secrete during an infection that causes major damage to the human body. These “acellular” (meaning not whole cell) vaccines also contain pertactin, fimbria, and filamentous hemagglutinin– all of the gook that pertussis produces –in a failed attempt to stop pertussis from sticking to itself and posting up in your throat. Pertussis and pertussis toxin are not the same thing— think of it like the difference between grapes and wine. Do you get arrested for driving after eating grapes? No, you don’t. Because although one is made from the other, they’re not the same thing.
Tylenol, including baby Tylenol, is one of the most widely used over-the-counter medications in the world. The same painkiller in baby Tylenol is sold under the brand “Calpol” in the United Kingdom and is commonly prescribed to babies to reduce fever and alleviate mild to moderate pain. Parents want to know how much to give to their baby. How much baby Tylenol is safe?
Baby Tylenol’s main ingredient is acetaminophen, known as paracetamol in Europe.
A nine-year joint research project conducted by VIB, KU Leuven and VUB has led to a crucial breakthrough in cancer research. Scientists have clarified how the Warburg effect, a phenomenon in which cancer cells rapidly break down sugars, stimulates tumor growth. This discovery provides evidence for a positive correlation between sugar and cancer, which may have far-reaching impacts on tailor-made diets for cancer patients. The research has been published in the leading academic journal Nature Communications.
This project was started in 2008 under the leadership of Johan Thevelein (VIB-KULeuven), Wim Versées (VIB-VUB) and Veerle Janssens (KU Leuven). Its main focus was the Warburg effect, or the observation that tumors convert significantly higher amounts of sugar into lactate compared to healthy tissues. As one of the most prominent features of cancer cells, this phenomenon has been extensively studied and even used to detect brain tumors, among other applications. But thus far, it has been unclear whether the effect is merely a symptom of cancer, or a cause.
Inflammation is a biochemical process resulting from pathogens, irritants, or damaged cells. It should not be compared to infection, although inflammation can result from infection. A cascade of biochemical events propagates and matures the inflammatory response, involving the local vascular system, the immune system, and various cells within the injured tissue. [Emphasis added]
Science teacher and author, Kent Heckenlively tells the story of how vaccines have become a thirty-year disaster since passage of the 1986 National Childhood Vaccine Injury Act.
More than 80 years ago, the renowned biochemist Otto Warburg described how cancer cells avidly consume glucose and produce lactic acid under aerobic conditions. Recent studies arguing that cancer cells benefit from this phenomenon, termed the Warburg effect, have renewed discussions about its exact role as cause, correlate, or facilitator of cancer. Molecular advances in this area may reveal tactics to exploit the cancer cell's “sweet tooth” for cancer therapy.
Yeast and cancer cells share the unusual characteristic of favoring fermentation of sugar over respiration. We now reveal an evolutionary conserved mechanism linking fermentation to activation of Ras, a major regulator of cell proliferation in yeast and mammalian cells, and prime proto-oncogene product. A yeast mutant (tps1∆) with overactive influx of glucose into glycolysis and hyperaccumulation of Fru1,6bisP, shows hyperactivation of Ras, which causes its glucose growth defect by triggering apoptosis. Fru1,6bisP is a potent activator of Ras in permeabilized yeast cells, likely acting through Cdc25. As in yeast, glucose triggers activation of Ras and its downstream targets MEK and ERK in mammalian cells. Biolayer interferometry measurements show that physiological concentrations of Fru1,6bisP stimulate dissociation of the pure Sos1/H-Ras complex. Thermal shift assay confirms direct binding to Sos1, the mammalian ortholog of Cdc25. Our results suggest that the Warburg effect creates a vicious cycle through Fru1,6bisP activation of Ras, by which enhanced fermentation stimulates oncogenic potency.
There's more bad news for sugar lovers.
After nine years of research, molecular biologists in Belgium have discovered that sugars stimulate tumor growth.
Their results, published Friday in the journal Nature Communications, help explain a puzzle oncologists have encountered for decades — and may offer some new, diet-based cancer-fighting solutions.
Exposes Ugly Truth Behind Vaccines, GMO's and Big Pharma.
Neutralizing antibodies have been thought to be required for protection against acutely cytopathic viruses, such as the neurotropic vesicular stomatitis virus (VSV). Utilizing mice that possess B cells but lack antibodies, we show here that survival upon subcutaneous (s.c.) VSV challenge was independent of neutralizing antibody production or cell-mediated adaptive immunity. However, B cells were absolutely required to provide lymphotoxin (LT) α1β2, which maintained a protective subcapsular sinus (SCS) macrophage phenotype within virus draining lymph nodes (LNs). Macrophages within the SCS of B cell-deficient LNs, or of mice that lack LTα1β2 selectively in B cells, displayed an aberrant phenotype, failed to replicate VSV, and therefore did not produce type I interferons, which were required to prevent fatal VSV invasion of intranodal nerves. Thus, although B cells are essential for survival during VSV infection, their contribution involves the provision of innate differentiation and maintenance signals to macrophages, rather than adaptive immune mechanisms.
The objectives of this study were to gather information regarding demographic and clinical characteristics of patients diagnosed with either fibromyalgia (FM) or chronic fatigue (CFS) following hepatitis B vaccination (HBVv) and furthermore to apply the recently suggested criteria of autoimmune (auto-inflammatory) syndromes induced by adjuvants (ASIA), in the aim of identifying common characteristics that may suggest an association between fibromyalgia, chronic fatigue and HBV vaccination.