In the case of flu shots given in pregnancy, there are two individuals to consider. If recommended, an influenza vaccine must be safe for both a mother and her unborn child. Many physicians and researchers question this purported safety...
Dr. Toni Bark gives a speech covering vaccines during pregnancy, particularly the TDaP and the MMR, and the miscarriage rate of women who receive them.
There is a very simple reason why the American College of Obstetricians and Gynecologists notes on their website that “no study to date has seen an adverse consequence of inactivate influenza vaccine in pregnant women and their offspring.” This is because no such study has been accurately designed and performed to determine the flu vaccine’s safety in pregnant women. The ACOG and CDC are not legally required to tell the truth about anything. They can spin words and science to their heart’s content without legal repercussions for wrong doing or criminal behavior. Nobody takes these associations and the federal agencies to court for cherry picking their studies or conducting remarkably poor clinical trials in order to promote their personal agendas and lies. However, private vaccine makers are required to provide the most accurate information available to vaccine administering physicians and healthcare workers on manufacturing package inserts. This is not only required to educate physicians about the vaccines being given to their patients, but also to provide a rough guideline in the event of vaccine adverse effects and injury. And what do we find on these inserts? Almost categorically, there is the warning that “safety and effectiveness have not been established in pregnant women or nursing mothers” for these flu vaccines.
There is a single question that needs to be answered: what is the actual gold standard proof to claim that the flu vaccine is safe for pregnant women and will not harm her fetus? There is none. Nor is there any valid evidence that flu vaccines are effective.
This webpage contains multiple videos and links to information about the dangers to to the fetus from vaccines given to a mother during pregnancy. The vaccines mentioned are the flu vaccine and DTaP.
The report by Anna Maria van Eijk and colleagues in The Lancet Infectious Diseases into coverage of interventions for malaria in pregnancy highlights the achievements and continuing challenges of global malaria control overall. The use of insecticide-treated nets by pregnant women and intermittent preventive treatment with sulfadoxine–pyrimethamine during pregnancy—shown to improve birth outcomes in endemic countries with moderate to high burden of disease2—are included in strategic plans for malaria programmes in nearly all endemic countries in sub-Saharan Africa.
Is there an association between maternal influenza infection and vaccination and autism risk?
In a cohort study of 196 929 children, of whom 3103 had austism spectrum disorder, maternal influenza infection during pregnancy was not associated with increased autism risk. There was a suggestion of increased risk of autism spectrum disorders among children whose mothers received an influenza vaccination during their first trimester, but the association was statistically insignificant after adjusting for multiple comparisons, indicating that the finding could be due to chance.
This video gives an overview to the risks associated with receiving vaccines when pregnant.
"Every pregnant mother should watch this video before going in for their first prenatal visit!!! You know those TVs in drs offices talking about recent studies, tips and tricks for illness and other news? If ONLY pregnant women could view this! Unfortunately they fall prey to what their OBS recommend despite lack of data on fetal toxicity.
There is plenty of data that shows the dangers of ingredients in vaccines and their potential to cause death and harm. WHY would they put the unborn at risk of death? "
Maternal immunization with tetanus toxoid, reduced diphtheria toxoid, and acellular pertussis (Tdap) vaccine could prevent infant pertussis.
This preliminary assessment did not find an increased risk of adverse events among women who received Tdap vaccine during pregnancy or their infants. For secondary outcomes, maternal immunization with Tdap resulted in high concentrations of pertussis antibodies in infants during the first 2 months of life and did not substantially alter infant responses to DTaP. Further research is needed to provide definitive evidence of the safety and efficacy of Tdap immunization during pregnancy.
Schizophrenia is perhaps the most enigmatic and tragic psychotic disorder with remarkable mortality and morbidity. Schizophrenia is complex and clinically a heterogeneous disorder. The etiological basis of schizophrenia ranges from autoimmune to neurodevelopmental hypothesis in one hand and involvement of different major gene segment with susceptibility loci on the other. Recently, neurodevelopmental hypothesis gained much impetus over the other domain. To support the neurodevelopmental basis, a number of investigations have shown that maternal infections during pregnancy increases the risk of the offspring developing schizophrenia and other neurodevelopmental disorders. The pathological mechanisms underlying this phenomenon is largely unknown. Many have suggested the involvement of different immune markers and shown that cytokines generated in response to maternal infection alter early brain development through their inflammatory activity. However, these findings have escaped discussion on various important issues related to cytokine homeostasis which depends on a large number of immune parameters including non-classical HLA-G molecules. Infections during early stages of pregnancy may alter cytokine regulation by disturbing the whole uterine immune milieu. To elucidate this issue, authors have tried to correlate the possible relationships between maternal infections and aberration of immune networking at the feto-maternal interface and their subsequent influence on the structural and functional abnormalities of the developing brain. The authors hypothesize that there exists a counter regulatory interaction among proinflammatory cytokines like TNF-α, HLA-G molecules and different immune cells like NK cells. We emphasize that HLA-G molecules are the novel immune players which maintain the immune homeostasis during early pregnancy in a manner that it can protect developing fetus from maternal immune attack. However, maternal infections may lead to the disturbance of HLA-G expression which in turn may fail to maintain its otherwise inhibitory potential to down regulate the detrimental inflammatory cytokines. Investigation on such interaction may unravel novel molecular mechanisms of neurodevelopmental basis of schizophrenia. Testing of our proposed hypothesis on animal models and on in vitro derived extravillous trophoblast cell lines holds promise of great insights to usher a new dimension of schizophrenia research and for developing new therapeutic strategies for better treatment and to adopt genetic prediction in schizophrenia management paradigm.
The requirement for vitamin A in reproduction was first recognized in the early
1900’s, and its importance in the eyes of developing embryos was realized shortly after.
A greater understanding of the large number of developmental processes that require
vitamin A emerged first from nutritional deficiency studies in rat embryos, and later from
genetic studies in mice. It is now generally believed that all-trans retinoic acid (RA) is the
form of vitamin A that supports both male and female reproduction as well as embryonic
development. This conclusion is based on the ability to reverse most reproductive and
developmental blocks found in vitamin A deficiency induced either by nutritional or
genetic means with RA, and the ability to recapitulate the majority of embryonic defects in
retinoic acid receptor compound null mutants. The activity of the catabolic CYP26
enzymes in determining what tissues have access to RA has emerged as a key regulatory
mechanism, and helps to explain why exogenous RA can rescue many vitamin A
deficiency defects. In severely vitamin A-deficient (VAD) female rats, reproduction fails
prior to implantation, whereas in VAD pregnant rats given small amounts of carotene or
supported on limiting quantities of RA early in organogenesis, embryos form but show a
collection of defects called the vitamin A deficiency syndrome or late vitamin A
deficiency. Vitamin A is also essential for the maintenance of the male genital tract and
spermatogenesis. Recent studies show that vitamin A participates in a signaling mechanism
to initiate meiosis in the female gonad during embryogenesis, and in the male gonad
postnatally. Both nutritional and genetic approaches are being used to elucidate the vitamin
A-dependent pathways upon which these processes depend.